A research team led by the National Institutes of Health has concluded that humans, like all other living things, are susceptible to the bacteria that cause tuberculosis.
The research was led by scientists at the University of Pennsylvania’s Perelman School of Medicine and conducted in collaboration with colleagues in India, the United Kingdom, Germany, Italy, Brazil, Canada, and the United States.
The researchers are reporting their findings in the February issue of the journal BioMed Central.
In their paper, the researchers concluded that the body’s natural defenses against tuberculosis are less developed in humans than in other species and may also be less efficient.
The body’s immune system is designed to protect against infection by invading bacteria, so a body that has a relatively weak immune system, such as humans, might have to fight the infection.
But in humans, the body has no way to clear out these invaders and they continue to grow and multiply.
Scientists are unsure how much of the immune system and immune function the body lost after people died of tuberculosis.
In the current study, the scientists examined data on the genomes of 1,200 people who had died from tuberculosis.
The scientists found that the genomes in the people who died of the disease had genetic variation that indicated they had been infected with tuberculosis.
They also looked at the genomes and proteins of more than 1,000 people who were alive and well at the time of death.
The people who lived in the study had the same genes as the dead people but had also been exposed to bacteria that the dead person had not.
These samples were then subjected to a series of tests to see if they had acquired the tuberculosis bacteria from the dead individual.
The researchers found that people who suffered the most from the tuberculosis were the people living with the highest levels of genetic variation.
Those with the most genetic variation were the most likely to have acquired the bacteria from someone who had lived with tuberculosis for years, the study authors wrote.
People who had had tuberculosis were more likely to be white, male, and had less education than people living in other ethnic groups.
These factors were also associated with a higher risk of acquiring the bacteria.
The study did not find a link between people with tuberculosis and those who had never developed the disease.
The authors noted that their study was limited to people who did not die of tuberculosis, which is not a common finding.
The findings have implications for treating the disease in humans.
They indicate that people with high levels of variation in the genes that are passed on to children may be more likely than people without genetic variations to be infected with the bacteria, the authors wrote, adding that the findings are important for other research into the biology of tuberculosis in humans and how to prevent or treat it.